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Chemistry World. Education in Chemistry. Open Access. Historical Collection.

You do not have JavaScript enabled. Please enable JavaScript to access the full features of the site or access our non-JavaScript page. Issue 13, Previous Article BG FM 003 Article.

From the journal: Journal of Materials Chemistry C. A new red phosphor BaGeF 6: You have access to this article.

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Please wait while we load your content Clin Sci. Increased plasma visfatin concentrations in morbidly obese subjects are reduced after gastric banding. The insulin-mimetic adipocytokine visfatin has been linked to obesity. The influence of weight loss on plasma visfatin concentrations in obese BG FM 003 is unknown yet. In this study we investigated whether plasma visfatin concentrations are altered by weight loss in patients with obesity.

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Visfatin plasma concentrations were markedly elevated in obese subjects BG FM 003. Insulin sensitivity as estimated by the homeostasis model assessment insulin resistance index was unchanged from 5. Elevated plasma visfatin concentrations in morbidly obese subjects are reduced after weight loss. This may be related to changes in insulin resistance over time. Plasma visfatin concentrations and fat depot-specific mRNA expression in humans.

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Visceral and subcutaneous adipose tissue display important metabolic differences that underlie the association of visceral obesity with obesity-related cardiovascular and metabolic alterations. Recently, visfatin was identified as an adipokine, which is predominantly secreted from visceral adipose tissue both in humans and mice. In this study, we examined whether visfatin plasma concentrations using BG FM 003 immunosorbent assay and mRNA expression using RT-PCR in visceral and subcutaneous fat correlates with anthropometric and metabolic parameters in subjects with a wide range of obesity, body fat distribution, insulin sensitivity, and glucose tolerance.

However, in a subgroup of 73 individuals, in which visceral fat mass was calculated from computed tomography scans, there was no correlation between plasma visfatin concentrations and visceral fat mass. We found no significant correlation between visfatin plasma concentrations and parameters of insulin sensitivity, including fasting insulin, fasting plasma glucose concentrations, and the glucose infusion rate during the steady state of BG FM 003 euglycemic-hyperinsulinemic clamp independent of percent body fat. Visfatin gene expression was not different between visceral and subcutaneous adipose tissue in the entire study group nor in selected subgroups.

In conclusion, visfatin plasma concentrations and visceral visfatin mRNA expression correlated with measures of obesity but not with visceral fat BG FM 003 or waist-to-hip ratio. In addition, we did not find differences in visfatin mRNA expression between visceral and subcutaneous adipose tissue in humans.

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How obesity causes diabetes: The BG FM 003 of obesity-associated diabetes is a major crisis in modern societies, in which food is plentiful and exercise is optional. The biological basis of this problem has been explored from evolutionary and mechanistic perspectives.

Evolutionary theories, focusing on the potential survival advantages of "thrifty" genes that are now maladaptive, are of great interest but are inherently speculative BG FM 003 difficult to prove. Mechanistic studies have revealed numerous fat-derived molecules and a link to inflammation that, together, are hypothesized to underlie the obesity-diabetes connection and thereby represent prospective targets for therapeutic intervention.

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